postsynaptic membrane वाक्य
उदाहरण वाक्य
मोबाइल
- This may make the postsynaptic membrane depolarized ( less negative ).
- These neurotransmitters bind to receptors on the postsynaptic membrane and lead to its depolarization.
- For example, curare is a poison that stops acetylcholine from depolarizing the postsynaptic membrane, causing paralysis.
- These inhibitory influences ( IPSPs ) are mediated by inhibitory neurotransmitter systems that cause postsynaptic membranes to hyperpolarize.
- Immediately behind the postsynaptic membrane is an elaborate complex of interlinked proteins called the postsynaptic density ( PSD ).
- Glutamate then binds to AMPA receptors contained in the postsynaptic membrane causing the influx of positively charged sodium atoms.
- When a glutamatergic synapse releases glutamate it binds to any AMPA and the NMDA receptors present in the postsynaptic membrane.
- Once in the synapse, neurotransmitters will rapidly move across the synapse to attach themselves to receptors on the postsynaptic membrane.
- When multiple EPSPs occur on a single patch of postsynaptic membrane, their combined effect is the sum of the individual EPSPs.
- The motor neuron then innervates the muscle fibers to contraction by causing an action potential on the postsynaptic membrane of the neuromuscular junction.
- The neurotransmitter molecules diffuse across the synaptic cleft to the postsynaptic membrane of the primary sensory neuron, where they elicit an action potential.
- Silent synapses release glutamate as do prototypical glutamatergic synapses, but their postsynaptic membranes contain only NMDA and possibly mGlu receptors able to bind glutamate.
- In the absence of an action potential, acetylcholine vesicles spontaneously leak into the neuromuscular junction and cause very small depolarizations in the postsynaptic membrane.
- The ectodomain then migrates across the synaptic cleft and binds to and activates a member of the EGF-receptor family on the postsynaptic membrane.
- Calcium ion diffusion into the presynaptic membrane signals the axon terminal to release quanta to generate either an IPSP or EPSP in the postsynaptic membrane.
- The drug blocks the active site of acetylcholinesterase so the enzyme can no longer break down the acetylcholine molecules before they reach the postsynaptic membrane receptors.
- The subsequent change in stimulation threshold of the postsynaptic membrane can be enhanced or inhibited, depending on the transmitter chemical involved and the ion permeabilities.
- Dok-7 is a postsynaptic protein that binds and activates MuSK protein, which then leads to AChR clustering and typical folding of the postsynaptic membrane.
- It may also play a role in ternary complex assembly of synaptic proteins at the postsynaptic membrane and coupling of signal transduction to membrane / cytoskeletal remodeling.
- These neurotransmitters are released into the synaptic cleft to bind onto the receptors of the postsynaptic membrane and influence another cell, either in an inhibitory or excitatory way.
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